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NEU.04 · Spinal Cord Injuries

Spinal cord injury,
revised the way the exam asks it.

The Blueprint lists no named conditions under this category, which means the whole category is fair game. Organised around the classification that drives everything, the function you can expect from each level, and the emergency you must never miss.

Foundations

Three questions answer almost every SCI case

What level, how complete, and how long ago. Get those three straight and the rest of the case largely writes itself, because they determine the function you expect, the complications you screen for, and the goals you set.

Question 01

What level?

The neurological level is the most caudal segment with normal sensory and at least antigravity motor function, with normal function above it. It is not the same as the level of the vertebral fracture, and examiners notice when candidates conflate the two.

Question 02

How complete?

Complete or incomplete, decided by sacral sparing, not by how much movement the person has. Incomplete injuries carry a materially better prognosis, which changes how you frame goals.

Question 03

How long ago?

Acute, subacute or chronic changes everything: spinal shock may still be masking the true picture, spinal stability and orthosis rules apply early, and the complications you screen for shift over time.

Traumatic and non-traumatic

Both belong in this category, and non-traumatic causes are common.

  • Traumatic: falls, motor vehicle collisions, sport, violence
  • Non-traumatic: tumour, infection such as abscess, transverse myelitis, vascular events, degenerative stenosis with myelopathy

Non-traumatic causes often present gradually, may be progressive, and may have no spinal instability at all, which changes precautions and prognosis.

Tetraplegia and paraplegia

  • Tetraplegia: cervical cord injury, affecting arms, trunk, legs and pelvic organs
  • Paraplegia: thoracic, lumbar or sacral injury, with arms spared

Above roughly T6 is the dividing line that matters most in practice, because that is where you gain the risk of autonomic dysreflexia and significant respiratory and blood pressure instability.

Upper motor neuron or lower motor neuron. Damage to the cord itself gives an upper motor neuron picture below the level, so expect spasticity, hyperreflexia and a reflexive bladder once spinal shock resolves. Damage at or below the conus and cauda equina gives a lower motor neuron picture, so expect flaccidity, areflexia and a flaccid bladder. This distinction drives your tone management, your bladder expectations and your prognosis.
Assessment and diagnosis

ISNCSCI: the language everyone on the team speaks

The International Standards for Neurological Classification of Spinal Cord Injury is how injuries are described everywhere. You do not need to be an examiner-level scorer, but you must understand what the letters mean and what drives them.

A Complete No motor or sensory function at S4 to S5. No sacral sparing B Sensory incomplete Sensation preserved below level, incl. S4-5. No motor below level C Motor incomplete Motor preserved below, most key muscles < 3. Not antigravity D Motor incomplete At least half of key muscles below are 3+. Antigravity or better E Normal Motor and sensory testing normal. Deficits may remain Everything hinges on whether S4 to S5 is spared. That is what separates A from B.

Sacral sparing, and why it decides everything

An injury is complete only when there is nothing at the very bottom of the cord. That is tested by:

  • Light touch and pin prick at the anal mucocutaneous junction, the S4 to S5 dermatome
  • Deep anal pressure
  • Voluntary anal contraction

If any of these is present, the injury is incomplete, regardless of how little the person can move. A person with almost no movement but intact sacral sensation is AIS B, not AIS A, and that matters for prognosis.

The zone of partial preservation describes segments below the level with some retained function in a complete injury.

Key muscles you should be able to name

LevelKey muscle action
C5Elbow flexors
C6Wrist extensors
C7Elbow extensors
C8Long finger flexors
T1Small finger abductors
L2Hip flexors
L3Knee extensors
L4Ankle dorsiflexors
L5Long toe extensors
S1Ankle plantarflexors

Confirm exact testing positions and grading against the current ISNCSCI worksheet before using clinically.

Do not classify during spinal shock. While spinal shock persists, reflexes are absent and the picture looks worse than it is. Completeness assessed too early can be wrong, and telling a family an injury is complete before the picture settles is a serious error. The initial examination is a baseline, not a prognosis.
Signature tool

Click a syndrome. Predict the presentation.

Incomplete injuries follow recognisable patterns depending on which part of the cord cross-section is damaged. Naming the syndrome and explaining the anatomy behind it is a reliable reasoning mark.

posterior (dorsal columns) anterior Cross-section. Schematic, not anatomical.

Tap a region, or use Tab plus Enter.

High yield

What function to expect from each level

This is the table examiners probe hardest, because it shows whether you can turn a classification into a realistic plan. Figures assume a motor complete injury with good rehabilitation and no major complications, and real people vary.

LevelKey muscles gainedRealistic functionMobility
C1–C3 Neck accessory muscles only Ventilator dependent. Total assistance for self care. Directs own care Power chair with chin, head or sip-and-puff control
C4 Diaphragm, upper trapezius May wean from ventilator. Total assistance for self care Power chair with chin or sip-and-puff control
C5 Deltoid, biceps, rhomboids Feeding, grooming and light tasks with adaptive equipment and set-up. Assistance for transfers Power chair. Manual chair short distances with rim adaptations
C6 Wrist extensors, serratus, pectoralis Tenodesis grasp. Upper body dressing, much of self care, assisted or independent board transfers Manual chair indoors. Driving with adaptations
C7–C8 Triceps, finger flexors and extensors Elbow extension changes everything. Independent transfers and pressure relief, independent self care Independent manual wheelchair, including many curbs
T1–T6 Hand intrinsics, upper intercostals Independent self care. Improving respiratory reserve. Still at risk of autonomic dysreflexia Independent manual wheelchair. Standing frame for bone and bowel health
T7–T12 Abdominals, lower intercostals Good trunk control, effective cough, independent in most activities Independent wheelchair. Exercise ambulation with orthoses possible but high energy cost
L1–L2 Hip flexors Independent. Household ambulation becomes realistic for some Ambulation with knee-ankle-foot orthoses and crutches, wheelchair for distance
L3–L4 Quadriceps Independent. Community ambulation realistic for many Ambulation with ankle-foot orthoses and a walking aid
L5–S1 Ankle dorsiflexors and plantarflexors Independent. Bladder, bowel and sexual function still commonly affected Functional ambulation, often with minimal or no orthosis

Tenodesis: protect it, do not stretch it away

With wrist extension preserved at C6, extending the wrist passively shortens the finger flexors and closes the hand. That passive grip is functional grasp for someone with no active finger flexion.

It depends on the long finger flexors staying slightly short. Aggressively stretching the fingers straight with the wrist extended can destroy the effect and cost the person their grip. Position and educate deliberately, and make sure everyone handling the hand knows.

Breathing tracks the level

  • C1–C2: diaphragm not innervated, ventilator dependent
  • C3–C5: phrenic nerve territory. The old line, the phrenic keeps the diaphragm alive, is worth remembering
  • Above T6: intercostals and abdominals impaired, so cough is weak and secretion clearance is a real risk
  • T7 and below: progressively better cough and respiratory reserve

Expect to use assisted cough, positioning and secretion clearance in cervical and high thoracic injuries. Respiratory complications are a leading cause of illness and death after cervical injury.

Domain C · Client safety and client-centred care

Autonomic dysreflexia: the one you cannot miss

If a spinal cord injury case appears in your oral exam, there is a strong chance this is the safety question. It is also a genuine emergency that can cause seizure, stroke or death, so learn it properly rather than as a list.

What it is. A noxious stimulus below the level of injury triggers an uncontrolled sympathetic response. Because descending inhibitory control cannot get past the lesion, vasoconstriction below the level continues unchecked and blood pressure climbs. The body compensates above the level only. It occurs in injuries at approximately T6 and above.
ABOVE the injury • Pounding headache • Flushed, red skin • Profuse sweating • Nasal congestion • Blurred vision • Anxiety, sense of dread Blood pressure rising Pulse often slow Compare to their own baseline BELOW the injury • Pale, cool skin • Goosebumps • Vasoconstriction Common triggers: 1. Bladder — blocked or kinked catheter, full 2. Bowel — impaction 3. Skin — pressure, ingrown nail, tight strap, burn 4. Fracture, infection, labour WHAT YOU DO 1. Stop the activity 2. SIT THEM UP Lower the legs. Never lie them flat. 3. Loosen everything Clothing, binder, straps 4. Find the cause Bladder first, then bowel 5. Monitor BP, escalate Stay with them. Call help.

Why you sit them up

This is the detail that separates a memorised list from understanding. Sitting upright and dropping the legs uses orthostatic pooling to bring blood pressure down. Lying the person flat, which is the instinct for someone who feels unwell, does the opposite and makes a dangerous blood pressure higher.

Say the reason out loud in an oral case. It demonstrates the reasoning rather than the recall.

Blood pressure in this population

Resting blood pressure is often low after a high spinal cord injury. A reading that looks unremarkable on paper can represent a substantial rise for that person.

Always compare to the individual's own baseline rather than to population norms, and treat a significant rise plus symptoms as dysreflexia until proven otherwise. Know their usual figures before you start a session.

Prevention is part of your job. Good bladder and bowel routines, pressure care, well-fitted equipment, avoiding tight straps and clothing, and educating the person and their family so they recognise it early. Many people with SCI carry a card describing dysreflexia management, and they are usually the expert in the room. Ask them.
Acute care

Spinal shock and neurogenic shock are not the same thing

Candidates mix these up constantly. One is a neurological state that resolves, the other is a cardiovascular emergency. Being able to separate them cleanly is an easy mark.

Neurological

Spinal shock

  • Temporary loss of all reflex activity below the level
  • Flaccid paralysis, areflexia, no bulbocavernosus reflex
  • Begins immediately after injury
  • Resolves over days to weeks, with return of reflexes signalling the end
  • Gives way to the upper motor neuron picture of spasticity and hyperreflexia

Implication: you cannot reliably classify completeness or give prognosis during this phase.

Cardiovascular

Neurogenic shock

  • Loss of sympathetic outflow, leaving vagal tone unopposed
  • Hypotension with bradycardia
  • Warm, dry, flushed peripheries, unlike other forms of shock
  • Associated with injuries above approximately T6
  • A medical emergency requiring haemodynamic management

Implication: expect blood pressure instability with position change, and coordinate closely with the medical team early on.

Orthostatic hypotension is your daily version of this. Sitting someone up for the first time after a high injury often drops their blood pressure sharply. Progress head-up position gradually, use compression stockings and an abdominal binder where indicated, monitor symptoms and blood pressure, and be ready to recline and elevate the legs. Notice that this is the opposite response to dysreflexia, which is exactly why you must tell them apart.
Domain A · Assessment and diagnosis

What you gather, and the order

Step 01

Chart and safety

  • Level, AIS grade, date of injury, mechanism
  • Spinal stability and orthosis orders, and any movement precautions
  • Surgical status and weight-bearing or mobilisation limits
  • Respiratory status, ventilation, secretion management
  • Baseline blood pressure, and any dysreflexia history
  • Bladder and bowel routine, skin integrity, current pressure areas
  • Pain, spasticity, medication
Step 02

Physical examination

  • Motor and sensory testing consistent with ISNCSCI
  • Tone and reflexes, and where the person is in spinal shock resolution
  • Respiratory assessment: breathing pattern, cough strength, auscultation
  • Range of motion, with tenodesis preserved in C6 and above
  • Skin inspection over pressure areas
  • Upper limb strength in detail, because it is the engine for everything
  • Orthostatic response to position change
Step 03

Function and participation

  • Bed mobility, rolling, supine to sit
  • Sitting balance, static and dynamic, and hand function during it
  • Transfers, and the level of assistance and equipment needed
  • Pressure relief: can they do it, how often, how effectively
  • Wheelchair set-up, propulsion and skills
  • Standing and walking where relevant, with orthoses
  • Home, work, school, transport, funding, and what the person wants
Evidence

Outcome measures worth naming

MeasureWhat it capturesWhy it fits here
ISNCSCI / AISNeurological level and completenessClassifies the injury itself and is the shared language of the team
SCIM III
Spinal Cord Independence Measure
Self care, respiration and sphincter management, mobilityThe SCI-specific functional measure. Name this one first
WISCI IIWalking ability with braces, aids and assistanceCaptures walking meaningfully when gait speed alone would not
10 Metre Walk Test / 6 Minute Walk TestGait speed and enduranceFor ambulatory incomplete injuries, tracks change over time
Berg Balance ScaleStanding balanceUseful in ambulatory incomplete injuries, floors in others
Wheelchair skills testingPropulsion, curbs, ramps, safe fallsThe mobility that actually determines community participation
Modified Ashworth ScaleResistance to passive movementTracks tone, but never treat tone in isolation from function
Neuropathic pain scalesCharacter and severity of painNeuropathic pain is common and drives quality of life

Confirm scoring and interpretation against the current source manual before relying on any measure clinically.

Domain B · Care planning

Restore, compensate, or both

Say which you are doing and why. In a complete injury you are largely building compensation and preserving the structures that make it possible. In an incomplete injury you also train for recovery. Most real plans do both at once.

The core of the plan

  • Upper limb strength and endurance. For most people with SCI the arms do the work of the legs, so shoulders, triceps and grip are functional priorities.
  • Sitting balance and trunk control, because everything else is built on it.
  • Transfer training, progressing assistance and technique deliberately.
  • Pressure relief training until it is automatic. This is not an add-on, it prevents a life-threatening complication.
  • Wheelchair skills, including propulsion technique, curbs, ramps and safe falling.
  • Respiratory care in cervical and high thoracic injuries: assisted cough, secretion clearance, inspiratory muscle work.
  • Range of motion and positioning, protecting tenodesis and preventing contracture.
  • Standing and weight bearing where appropriate, for bone health, tone, bowel function and wellbeing.
  • Activity-based therapy and gait training in incomplete injuries, including body weight supported treadmill training where available.
  • Cardiovascular fitness, because cardiometabolic disease is a major long-term risk.

Protect the shoulders, from day one

Shoulder pain is extremely common in long-term wheelchair users, and losing shoulder function costs independence in a way losing it elsewhere does not.

  • Train posterior shoulder and scapular stabilisers, not just the pushing muscles
  • Coach efficient propulsion: long, smooth strokes, low cadence, hand dropping below the rim on recovery
  • Optimise wheelchair set-up, since axle position and seat height change shoulder load
  • Vary transfer technique and direction where possible
  • Stretch anterior structures, strengthen posterior ones
Goal setting. Written with the person, measurable, and anchored to participation. People with SCI are often young, and goals about work, study, driving, sport, parenting and relationships are legitimate physiotherapy territory, not extras.
Clinical management

Complications you are expected to prevent

Early and ongoing

  • Pressure injury. The most preventable and most damaging. Turning schedules, cushion and mattress prescription, daily skin checks, pressure relief every few minutes in sitting, and education. Insensate skin does not warn anyone.
  • Deep vein thrombosis and pulmonary embolism. Risk is high early. Know the prophylaxis, watch for calf signs, and escalate breathlessness or chest pain.
  • Respiratory complications. Weak cough leads to retained secretions, atelectasis and pneumonia. Assisted cough and positioning are core physiotherapy.
  • Orthostatic hypotension. Progress upright position gradually with compression and binders.
  • Contracture. Range of motion and positioning, while deliberately preserving tenodesis.

Later and long-term

  • Spasticity. Sometimes useful for transfers and standing, sometimes disabling. Treat function, not tone scores. A sudden increase can signal infection, pressure injury or syringomyelia.
  • Heterotopic ossification. Bone forming in soft tissue, often around hips and knees, presenting with warmth, swelling and reducing range. Escalate rather than force through it.
  • Neuropathic pain. Burning, shooting pain at or below the level. Does not respond to a musculoskeletal approach and needs medical management.
  • Osteoporosis and fragility fracture below the level of injury. Handle limbs with care during transfers and range of motion.
  • Syringomyelia. A late cyst in the cord causing ascending sensory loss, new weakness, increasing pain or changing tone. New neurological change in a stable person always warrants referral.
  • Cardiometabolic disease and reduced bone density, driven by reduced activity. Fitness is preventive medicine here.
Domain E and F · Collaboration and communication

Who you refer to, and exactly why

Occupational therapy

Upper limb function, self care, seating and pressure cushion prescription, home and vehicle modification, assistive technology, return to work and study.

Nursing and physician

Bladder and bowel programmes, skin integrity, spasticity and pain management, autonomic dysreflexia protocols, medical stability and clearance.

Respiratory therapy

Ventilation, weaning, cough assist devices and secretion clearance in cervical and high thoracic injuries.

Psychology and peer support

Adjustment, mood, and identity after a life-changing injury. Peer mentors with lived experience often achieve what clinicians cannot.

Social work and vocational

Funding, equipment approval, housing, attendant care, return to work or school, and family and carer support.

Dietitian and specialist clinics

Nutrition for wound healing and weight management, plus specialist referral for sexual health, fertility, and urology.

Communication note. Talk to the person, not the attendant or family member pushing the chair. Get to eye level for a conversation rather than standing over a seated person. Ask before touching a wheelchair, because it is part of their personal space, not equipment lying around. And remember that a person living with SCI for years usually knows their own body, routines and dysreflexia triggers better than you do, so ask them first.
Domain D and G · Professional responsibilities and practice management

Prognosis, scope, and the conversations people actually want

Situations that show up in cases

  • "Will I walk again?" Do not promise, and do not flatly refuse hope. Explain that prognosis depends on level and completeness, that the picture is clearer once spinal shock resolves, that most recovery happens in the first months while gains continue after, and that the team will keep reassessing. Involve the physician for the prognostic discussion. Then redirect to what you can influence now.
  • Questions about sex and fertility. Common, important, and often never asked because no one opens the door. Acknowledge the question seriously and refer to the appropriate specialist service rather than deflecting.
  • Refusing equipment such as a wheelchair or cushion. Explore the reason, which is usually identity, provide clear information about the risk, respect the decision, document it, and revisit.
  • Driving and return to work. Report functional findings, refer for driving assessment, and direct licensing questions to the physician and provincial authority.
  • Outside your competence. Say so and seek supervision. Specialist SCI centres exist because this is a specialist area.

Consent, dignity and documentation

  • Capacity is presumed. A ventilated person who communicates by mouthing or a device is still the decision maker.
  • Consent is ongoing, and physically dependent people can find it hard to refuse. Check in explicitly and give real opportunities to decline.
  • Privacy and dignity during skin checks, bowel routines and transfers. Ask who the person wants present.
  • Document objectively, including baseline blood pressure, skin findings, assistance levels, and any dysreflexia episode with its trigger and response.
  • Assigning to support personnel is acceptable for stable repetitive tasks with supervision, never for assessment, interpretation, or the decision to progress.
Oral section practice

Answer out loud, then reveal

Speak for sixty seconds before opening each card.

During a sitting balance session your client with a T4 AIS A injury develops a sudden pounding headache, is flushed and sweating above the chest, and looks anxious. What is happening and what do you do?

Recognise it. This is autonomic dysreflexia until proven otherwise. The injury is above T6, and the pattern of pounding headache with flushing and sweating above the level fits.

Act in order. Stop the activity. Sit the client upright and lower their legs, because orthostatic pooling helps bring blood pressure down, and lying them flat would make it worse. Loosen tight clothing, the abdominal binder and any leg bag straps. Measure blood pressure and compare it against their own baseline, which is often low.

Find the cause. Check the bladder first, looking for a blocked or kinked catheter or a full bag, because bladder causes are the most common. Then consider bowel impaction, and then skin, including pressure, tight straps or an ingrown nail.

Escalate. Call for help and stay with the client. Monitor blood pressure frequently. If it does not settle quickly this is a medical emergency requiring urgent medical management, because it can cause seizure, stroke or death. Document the episode, the trigger and the response.

Explain the difference between spinal shock and neurogenic shock, and why it matters to you.

Spinal shock is a temporary loss of reflex activity below the level of injury. The person is flaccid and areflexic, and it resolves over days to weeks as reflexes return, giving way to the upper motor neuron picture of spasticity and hyperreflexia.

Neurogenic shock is haemodynamic. Loss of sympathetic outflow leaves vagal tone unopposed, producing hypotension with bradycardia and warm, dry peripheries, and it is associated with injuries above roughly T6. It is a medical emergency.

Why it matters to me. I cannot reliably classify completeness or discuss prognosis during spinal shock, so I treat early findings as a baseline rather than a forecast. And in neurogenic shock I expect blood pressure instability with any position change, so I progress upright positioning gradually, monitor closely, and coordinate with the medical team.

Your client has a C6 AIS A injury. What functional outcomes are realistic, and what must you protect during your treatment?

Realistic function. C6 preserves wrist extension. With good rehabilitation I would expect independence or near independence in upper body dressing and much of self care with equipment, transfers with a board ranging from assisted to independent, manual wheelchair use particularly indoors, and driving with adaptations. Lower body dressing and some transfers may still need assistance.

What I protect. Tenodesis grasp. Extending the wrist passively shortens the long finger flexors and closes the hand, giving functional grip without active finger flexion. That depends on those tendons staying slightly short, so I would avoid stretching the fingers straight with the wrist extended, position the hand deliberately, and educate the client, family and all staff handling the hand.

The rest of the plan. Upper limb strengthening with attention to posterior shoulder and scapular stabilisers, sitting balance, transfer training, pressure relief until it is automatic, wheelchair skills, and respiratory care given the level. Plus dysreflexia education, because C6 is above T6.

A client asks you directly, on day four after a cervical injury, whether they will ever walk again. How do you respond?

Do not answer with a prediction. At day four the client may still be in spinal shock, so the neurological picture is not settled and any classification is provisional.

Be honest and humane. I would acknowledge how important the question is, explain that it is genuinely too early to answer accurately, describe what determines the answer, which is the level and completeness of the injury, and explain that we reassess formally as the picture becomes clearer. I would say that most neurological recovery happens in the first months while improvement continues beyond that.

Redirect to what is controllable. Describe the plan for the coming weeks and the short-term goals we can work on now, and make clear I am not avoiding the question but answering it as honestly as I can.

Involve the team. Flag the conversation to the physician and the team so the prognostic discussion is consistent, and offer psychology and peer support. Document what was discussed.

What loses marks. Promising recovery, flatly saying never, or deflecting with "you should ask your doctor" and nothing else.

Your client with a T10 paraplegia has a stage 2 pressure injury over the ischial tuberosity. How does this change your management?

Offload first. The priority is removing pressure from the area, which means reviewing sitting tolerance and possibly restricting sitting time, in discussion with nursing and the team. Wound care is nursing and medical territory but the offloading plan is very much mine.

Find out why it happened. Review the cushion and its age and fit, wheelchair set-up and posture, transfer technique that may be dragging over the tuberosity, pressure relief frequency and effectiveness, moisture, nutrition and any recent change in routine or equipment.

Retrain and re-equip. Reinforce pressure relief technique and frequency until it is automatic, correct transfer technique to lift rather than drag, and refer to occupational therapy for cushion and seating reassessment.

Keep the rest going. Continue strengthening, respiratory and fitness work in positions that do not load the area, so the person does not lose conditioning while healing. Educate on daily skin checks, and document the plan and the review date.

A colleague suggests aggressively stretching your C6 client's fingers into full extension to prevent contracture. How do you respond?

I would explain the reasoning rather than just disagreeing. In a C6 injury there is no active finger flexion, and functional grip depends on tenodesis, where passive wrist extension shortens the long finger flexors and closes the hand.

That effect relies on those tendons remaining slightly short. Stretching the fingers into full extension with the wrist extended can lengthen them and remove the client's only functional grasp, which would be a real loss of independence.

So the goal is not maximum range, it is preserving the specific tissue length that makes grip work. I would maintain range in a controlled way, avoid simultaneous full wrist and finger extension, use appropriate positioning or splinting, and make sure everyone handling the hand, including family and support staff, understands why. I would document the precaution clearly.

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